Vitrectomy Eye Surgery: What to Expect Before, During and After

Vitrectomy Eye Surgery What You Need to Know Before, During and After

Vitrectomy Eye Surgery What You Need to Know Before, During and After

Reviewed by Dr Wilson Wong Jun Jie, Vitreoretinal and Refractive Surgeon of OasisEye Specialists | Last updated: August 2026



Quick answers

What is it?  Vitrectomy (pars plana vitrectomy, PPV) is microsurgery to remove the vitreous gel from inside the eye, giving the surgeon direct access to treat the retina.

Who needs it?  Patients with retinal detachment, macular hole, vitreous haemorrhage, epiretinal membrane, or diabetic eye disease causing vision loss.

How long does it take?  45 minutes to 2 hours for most cases; combined phaco-vitrectomy or complex retinal work may run longer.

How long is recovery?  Activity restrictions last 4–6 weeks. Full visual recovery takes 3–6 months, especially if the macula was involved or silicone oil was used.

Can I fly afterward?  Not while a gas bubble is in your eye — typically a 3–8 week ban depending on gas type. Silicone oil carries no flying restriction.

 

Key facts about vitrectomy

Vitrectomy is a common eye operation to treat retinal problems. Surgeons remove the vitreous gel and replace it with gas or silicone oil to help the retina heal. Most patients go home the same day.

The operation takes one to two hours. Recovery takes four to six weeks for basic activities. Most patients need to avoid flying until their gas bubble has gone.

 

What Is Vitrectomy Surgery, and Who Performs It?

Vitrectomy — formally pars plana vitrectomy (PPV) — is a microsurgical procedure in which a vitreoretinal surgeon removes the vitreous gel from the back of the eye to treat retinal diseases. It is performed as day surgery under local or general anesthesia in 45 minutes to 2 hours.

The vitreous is the clear, gel-like substance that fills the eye between the lens and the retina. When disease affects the retina — through bleeding, traction, or detachment — the vitreous must be removed to allow safe surgical access. Modern microincision vitrectomy surgery (MIVS) uses instruments as small as 27-gauge (0.4 mm) inserted through the pars plana, a thin region of the eye wall 3–4 mm behind the limbus. 

 

Who Needs a Vitrectomy? Conditions Treated

Vitrectomy is used to treat retinal detachment, macular hole, epiretinal membrane (macular pucker), vitreous haemorrhage from diabetic eye disease, dropped lens nucleus during cataract surgery, vitreous floaters, myopic traction maculopathy, and proliferative vitreoretinopathy (PVR).

Your vitreoretinal surgeon may recommend vitrectomy for any of the following:

  • Retinal detachmentthe retina separates from the back of the eye, threatening permanent vision loss if untreated within hours to days.
  • Macular holea full-thickness break in the central retina causing blurred or missing central vision.
  • Epiretinal membrane (macular pucker) — scar tissue on the retinal surface causing visual distortion and reduced acuity.
  • Vitreous haemorrhage — bleeding into the vitreous cavity, most commonly from proliferative diabetic retinopathy (PDR) or a retinal tear.
  • Diabetic tractional retinal detachment (TRD) – in advanced proliferative diabetic retinopathy, fibrovascular membranes contract and mechanically pull the retina away from the back of the eye. Vitrectomy releases this traction, removes the membranes, and allows laser treatment of the ischaemic retina. Surgery is typically combined with anti-VEGF injection 3-5 days beforehand to reduce intraoperative
  • Proliferative vitreoretinopathy (PVR) — scar tissue on both sides of the retina, often complicating retinal detachment repair.
  • Dropped nucleus — a lens fragment falling into the vitreous during cataract surgery.
  • Vitreous floaters (floaterectomy) — in patients with dense, vision-degrading floaters where conservative management has failed.
  • Myopic traction maculopathy (MTM) — vitreoretinal traction distorting or detaching the macula in highly myopic eyes, sometimes combined with a macular buckle.

 

What Happens During a Vitrectomy? A Step-by-Step Overview

Vitrectomy is performed as day surgery under local or general anaesthesia. The surgeon makes three micro-incisions in the sclera, removes the vitreous with a high-speed cutter, treats the retina directly, and fills the eye with a tamponade agent — air, gas, or silicone oil — before closing.

Step 1 — Anaesthesia

Most vitrectomies in Malaysia are performed under local anaesthesia with sedation: a peribulbar or sub-Tenon’s block numbs the eye completely while sedation keeps you comfortable. General anaesthesia is reserved for children, complex cases, or patients who cannot remain still.

Step 2 — Creating access

Three micro-incisions are made in the sclera (the white of the eye) at the pars plana, typically using 23-gauge (0.6 mm), 25-gauge (0.5 mm), or 27-gauge (0.4 mm) instruments. One port carries the infusion line (balanced saline solution, BSS), one carries the light fibre, and one carries the vitreous cutter.

Step 3 — Removing the vitreous

The vitreous cutter oscillates at up to 10,000 cuts per minute, aspirating the vitreous gel in fine fragments. BSS flows simultaneously through the infusion cannula to maintain eye pressure and shape throughout.

Step 4 — Treating the retina

With the vitreous cleared, the surgeon addresses the underlying problem: peeling an epiretinal membrane or internal limiting membrane (ILM) using fine forceps stained with chromovitrectomy dyes (brilliant blue G or triamcinolone), applying endolaser to seal retinal breaks, draining subretinal fluid, or mechanically unfolding and reattaching a detached retina.

Step 5 — Tamponade: filling the eye

The vitreous cavity is filled with a tamponade agent that holds the retina in place while it heals. The choice depends on the condition treated, expected healing time, and your ability to position your head after surgery.

TamponadeTypeDurationFlying banNotes
AirGas5–10 days~10 daysShort tamponade; simpler cases
SF6 (20%)Gas2–3 weeks~3 weeksMost common for macular holes
C2F6 (16%)Gas4–5 weeks~6 weeksMedium duration tamponade
C3F8 (14%)Gas6–8 weeks~8 weeksLongest-acting intraocular gas
Silicone oilOilUntil surgically removedNo restrictionComplex detachments; requires 2nd procedure

 

Vitrectomy Combined with Cataract Surgery — Should You Have Both at Once?

Combined phaco-vitrectomy — cataract removal and vitrectomy in the same operation — is recommended when an existing cataract clouds the surgeon’s view, or when vitrectomy would otherwise cause a cataract within 1–2 years. One operation means one anaesthetic, one recovery period, and lower total cost.

Vitrectomy significantly accelerates cataract progression: most patients develop a visually significant cataract within 1–2 years of vitrectomy. 

 

Vitrectomy Recovery: A Week-by-Week Timeline

Recovery after vitrectomy involves activity restrictions for 4–6 weeks, with most patients returning to light work in 1–2 weeks. Full visual recovery takes 3–6 months. The exact timeline depends on the tamponade used and whether the macula was involved.

TimeframeWhat happensWhat you can do
Day 1–3Eye patch off, antibiotic + steroid drops begin, first reviewRest, no bending, no lifting
Week 1Positioning (if gas), blurred vision expected, follow-up visitNo driving, no flying, no strenuous activity
Weeks 2–4Gas bubble shrinking, vision gradually improves top-down
Light desk work possible from Week 2
Month 1–3
Gas fully absorbed (SF6 by Week 3, C3F8 by Week 8)
Driving resumes once VA adequate; glasses review
Month 3–6Full visual recovery; silicone oil removal if applicable
Most patients back to normal activity

 

Activity restrictions — numbered checklist

Follow these in order of importance during your gas tamponade period:

  1. Do not fly or travel to high altitude while gas is present — gas expands with reduced atmospheric pressure and can dangerously raise eye pressure.
  2. Do not drive until your surgeon confirms the gas has cleared and your vision in both eyes is adequate.
  3. Maintain prescribed head positioning for the specified hours per day — face-down, side-lying, or upright depending on your condition and as advised by the surgeon. Positioning compliance directly affects surgical success.
  4. Avoid strenuous activity, heavy lifting, and bending at the waist for at least 4–6 weeks.
  5. Avoid swimming, pools, and sea water for at least 6 weeks to reduce infection risk.
  6. Instill antibiotic and steroid eye drops exactly as prescribed, typically for 4–6 weeks.

Clinical note: Positioning compliance is one of the strongest modifiable predictors of anatomic success in macular hole surgery. Studies report closure rates above 95% with full compliance, falling to 75–80% with partial compliance (Steel & Lotery, Eye 2013).

 

Will My Eye Collapse If the Vitreous Is Removed?

No. The eye does not collapse after vitrectomy. The tamponade agent — air, gas, or silicone oil — temporarily maintains the eye’s shape and pressure. As gas dissolves, the eye refills naturally with aqueous humour, a clear fluid produced continuously by the ciliary body.

Eye pressure is measured at every post-operative visit. A transient rise is possible with gas or silicone oil tamponade, which is why your drops schedule and follow-up visits are essential even if your eye feels comfortable.

 

Vitrectomy vs Scleral Buckle — What Is the Difference?

A scleral buckle indents the eye wall from the outside to relieve retinal traction — no internal entry required. Vitrectomy works from the inside, directly releasing traction and reattaching the retina. Buckles are preferred for young myopic patients with simple peripheral detachments; vitrectomy suits posterior breaks, PVR, and complex cases.

The two procedures are not mutually exclusive — combined vitrectomy and scleral buckle (the ‘360-degree buckle plus PPV’) is used for the most complex or recurrent detachments to maximise anatomical success.

Anatomic success rates: Primary vitrectomy achieves retinal reattachment in over 90% of cases for most indications (American Society of Retina Specialists, 2023). In centres of excellence, single-surgery success for primary retinal detachment exceeds 85–90% (Heimann et al., Ophthalmology 2007).

Frequently Asked Questions About Vitrectomy

These are the questions patients most commonly ask before and after vitrectomy surgery in Malaysia, together with direct answers.

Most vitrectomies take 45 minutes to 2 hours. Combined phaco-vitrectomy or cases requiring membrane peeling, laser, and fluid-gas exchange may take longer. Your surgeon will give a specific estimate at your pre-operative consultation.

Vitrectomy is not painful during the procedure — the eye is fully numbed with a peribulbar or sub-Tenon’s block. Afterward, most patients describe only a mild gritty or pressure sensation. Significant pain is uncommon and should be reported promptly, as it may signal raised eye pressure.

Vitrectomy is highly effective. According to the American Society of Retina Specialists (ASRS), anatomic success — the retina remaining properly reattached — exceeds 90% for most conditions. Visual outcome depends on whether the macula was involved and how long the retina was detached before surgery.

Key statistic: Anatomic success after vitrectomy for primary retinal detachment exceeds 90% in experienced hands. Macular hole closure rates using SF6 gas and ILM peeling exceed 90–95% (AAO Preferred Practice Pattern, 2023).

No — not while a gas bubble is in your eye. Reduced atmospheric pressure at altitude causes the gas to expand, which can spike eye pressure dangerously. Flying is safe once gas has fully dissipated: approximately 10 days for air, 3 weeks for SF6, 6 weeks for C2F6, and 8 weeks for C3F8. Silicone oil carries no flying restriction.

You cannot drive while a gas bubble is present or while your operated eye has substantially reduced vision. Most patients with gas tamponade can resume driving at 6–8 weeks, once the bubble has cleared and visual acuity is adequate in both eyes. Your surgeon confirms this at a follow-up visit.

Activity restrictions — no flying, no driving, positioning, no strenuous exercise — last 4–6 weeks. Full visual recovery takes 3–6 months, particularly if the macula was affected or silicone oil was used. Follow-up visits are typically at Day 1, Week 1, Month 1, and Month 3.

At private ophthalmology centres in Kuala Lumpur, vitrectomy surgical fees range from approximately RM 12,000 to RM 18,000, depending on complexity, tamponade used, and whether cataract surgery is combined. This excludes anaesthesia, facility, and post-operative care. Medical insurance coverage varies — confirm with your insurer before surgery.

Vitrectomy is safe in experienced hands, but risks include: cataract formation in phakic eyes (very common, within 1–2 years), raised intraocular pressure with gas or oil tamponade, re-detachment (~5–10%), endophthalmitis (serious infection, ~0.03–0.05%), and macular oedema (uncommon). Your surgeon will discuss the specific risk profile for your case.

Some patients require updated glasses once vision stabilizes at the 3-month mark. If silicone oil was used, a second procedure to remove it is typically planned 3–6 months after the initial surgery, once the retina has healed. Oil removal is a short, well-tolerated operation.

Laser vitreolysis uses a YAG laser to fragment vitreous floaters without surgery, but its effectiveness is limited to specific floater types — it is not suitable for dense or posterior floaters. Floaterectomy (vitrectomy) removes floaters more completely but carries the risks of any intraocular surgery. Your surgeon will advise which approach suits your floaters. OasisEye Specialists does not offer Laser vitreolysis.

When to See a Vitreoretinal Surgeon — Urgent Warning Signs

Seek same-day urgent assessment if you notice a sudden shower of new floaters, flashing lights, a shadow or curtain across your vision, or sudden painless loss of vision. These are signs of a retinal tear or detachment — a time-sensitive emergency where every hour matters.

Do not wait for a routine appointment. Retinal detachment extending to the macula causes permanent central vision loss within 24–72 hours of macular involvement. Early assessment gives you the best chance of preserving central vision.

Urgent symptoms requiring same-day review:

  • Sudden shower of new floaters, especially with flashing lights
  • A dark shadow, curtain, or veil across part of your visual field
  • Sudden, painless loss of central or peripheral vision
  • Blurred vision that does not resolve within 24 hours


References and Further Reading

  1. American Society of Retina Specialists (ASRS). Retinal Detachment patient information. asrs.org. Accessed July 2026.
  2. American Academy of Ophthalmology. Preferred Practice Pattern: Posterior Vitreous Detachment, Retinal Breaks and Lattice Degeneration. aao.org. 2023.
  3. Heimann H, et al. Scleral buckling versus primary vitrectomy in rhegmatogenous retinal detachment. Ophthalmology. 2007;114(12):2142–2154.
  4. Steel DHW, Lotery AJ. Idiopathic vitreomacular traction and macular hole: a comprehensive review of pathophysiology, diagnosis, and treatment. Eye. 2013;27(Suppl 1):S1–S21.
  5. Wong CW, et al. Myopic traction maculopathy: an update. Clinical and Experimental Ophthalmology. 2015;43(9):838–848.
  6. Bhende M, et al. Vitrectomy for vitreous floaters: outcomes and patient satisfaction. Eye. 2013.
  7. Stalmans P, et al. Enzymatic vitreolysis with ocriplasmin for vitreomacular traction and macular holes. NEJM. 2012;367:606–615.


This article is for general informational purposes only and does not constitute medical advice, diagnosis, or treatment. Always consult a qualified vitreoretinal surgeon about your specific condition. Clinical details may vary depending on individual patient factors, surgeon technique, and available equipment.

Retinopathy of Prematurity (ROP): A Comprehensive Guide for Parents and Caregivers

Retinopathy of Prematurity (ROP) A Comprehensive Guide for Parents and Caregivers

Retinopathy of Prematurity (ROP) A Comprehensive Guide for Parents and Caregivers

Reviewed by Dr Manoharan Shunmugam, Vitreoretinal Surgeon of OasisEye Specialists | Last updated: July 2026



As a vitreoretinal specialist, few conditions demand earlier attention – or carry higher stakes – than
Retinopathy of Prematurity (ROP). Every week in our clinics across Malaysia, we meet families who had never heard of ROP before their baby arrived early. This article explains everything you need to know: what ROP is, why it happens, how it is staged, what treatment can offer, and – most importantly, what you as a parent can do to protect your child’s vision.

 

What is Retinopathy of Prematurity?

Retinopathy of Prematurity (ROP) is a leading, yet preventable, cause of childhood blindness. When infants are born too early, incomplete blood vessel growth in the retina can trigger abnormal vessel development, potentially leading to scarring, retinal detachment, and permanent vision loss. Because early detection is critical to saving a child’s sight, our team is committed to rigorous screening protocols.

 

What Causes ROP?

The root cause is preterm birth itself, but several factors can compound:

  • Oxygen levels: Your neonatologist will be able to advise you on the optimal oxygen level for your baby. Sometimes in spite of this, the normal signalling that guides blood vessel growth in the retina doesn’t occur and ROP develops.
  • Systemic inflammation: Infection and inflammatory processes in a premature baby’s body can further damage fragile retinal vessels.
  • Sepsis and intraventricular haemorrhage: These serious complications of prematurity are associated with an increased risk of developing ROP.
  • Blood transfusions: Repeated blood transfusions may be required if haemoglobin levels are low, and as a result of this, altered oxygen-carrying capacity, blood vessel development is also affected.
  • Poor postnatal weight gain: Growth velocity after birth is a recognised marker of ROP severity – infants who gain weight slowly may require more regular eye checks to monitor ROP status.
  • Nutritional status and lung maturity: Both affect how effectively the body regulates the metabolic environment of the developing eye.

 

The 5 Stages of ROP: What Each One Means

Stage 1 – Mild

A faint demarcation line separated normal from abnormal retinal tissue. Most infants at this stage improve spontaneously without any treatment.

Stage 2 – Moderate

The demarcation line develops into a raised ridge. Spontaneous regression is still common, but close monitoring is essential.

Stage 3 – Severe

Abnormal blood vessels grow from the ridge into the vitreous gel. This is the stage at which many infants require treatment.

Stage 4 – Partial Retinal Detachment

Scar tissue begins to pull the retina away from the back of the eye. Surgical intervention is necessary, though visual outcomes are more guarded.

Stage 5 – Total Retinal Detachment

The retina is completely detached. Even with surgery, some degree of permanent visual impairment is likely. This underscores why early detection is so critical – the vase majority of the Stage 5 cases are preventable through timely screening. 

 

Signs and Symptoms of ROP: What Parents Should Watch For

Early- stage ROP is invisible to the naked eye and can only be caught through dilated eye exams by an ophthalmologist. However, parents should watch for these signs of advanced disease:

  • White pupil glow (leukocoria): A whitish reflection in photos or dim light. This is an urgent warning sign.
  • Abnormal eye movements: Though this may be present in normally developing eyes within the first few weeks after birth, if your child’s eyes are turning in/out or shaking involuntarily and you are concerned, please get this checked by an Ophthalmologist.
  • Lack of visual responsiveness: Failing to track objects, fixate on faces, or respond to light by a few months of corrected age. 

If you notice any of these signs, contact a paediatric eye specialist immediately – do not wait for your next appointment.

 

Treatment Options for ROP

When ROP reaches a threshold requiring intervention, our team has several effective tools at our disposal. The choice of treatment depends on the stage of ROP, its severity and the individual clinical picture.

Laser Therapy (Laser Photocoagulation)

Laser photocoagulation is the long-standing and effective treatment for RIOP. Performed under local or general anaesthesia, it uses precise laser burns to ablate the unhealthy peripheral retina. This eliminates the tissue sending out abnormal growth signals, effectively stopping the disease.

Anti-VEGF Injections

A major recent advance in ROP management is anti-VEGF injections, which block the chemical signal driving abnormal vessel growth. They are especially valuable for aggressive or hard-to-reach cases and are often combined with laser therapy.

While an eye injection sounds daunting to parents, the procedure takes only seconds, uses local anaesthesia, and is well-tolerated. However, because ROP can sometimes recur, close and extended follow-up is essential following this treatment. 

Vitreoretinal Surgery (Stages 4-5)

When a retinal detachment has occurred, surgical intervention by an experienced vitreoretinal surgeon offers the best chance of preserving some useful vision. Options include vitrectomy (removal of the vitreous gel and scar tissue) and scleral buckling (placing a silicone band around the eye to support the retina). Outcomes at Stages 4-5 are more variable, and some degree of permanent visual impairment may remain – reinforcing once again that prevention and early treatment are paramount.

 

Screening and Prevention: The Foundation of ROP Care

The most powerful tool we have against ROP is timely screening. According to the Ministry of Health Malaysia/s latest Clinical Practice Guidelines (2023), retinal examinations are recommended for infants who meet any of the following criteria:

  • Gestational age less than 34 weeks
  • Birth weight less than 1,750 g
  • An unstable clinical course that puts them at high risk, as determined by the attending neonatologist or paediatrician (regardless of gestational age or birth weight)

Beyond the neonatal period, children who had significant ROP – even those treated successfully – require ongoing eye follow-up throughout childhood. Myopia (short-sightedness), amblyopia (lazy eye), strabismus, and glaucoma are all more common in this population, and early management of these conditions gives children the best possible visual outcomes as they grow.

Parents of premature infants should advocate for themselves: if your baby meets the screening criteria and has not yet been referred to an ophthalmologist, ask your neonatologist or paediatrician to arrange this as a matter of urgency.

Frequently Asked Questions About ROP

Infants born before 34 weeks gestation or with a birth weight below 1,750 grams carry the highest risk, as defined by the Ministry of Health Malaysia’s 2023 Clinical Practice Guidelines. Risk is further increased in babies who experience sepsis, intraventricular haemorrhage, repeated blood transfusions, prolonged supplemental oxygen, or slow postnatal weight gain. Your neonatologist will identify high-risk infants early and arrange the appropriate ophthalmology referral.

ROP is diagnosed through a dilated eye examination performed by an ophthalmologist using a specialised lens and indirect light source. Dilating drops are applied first to widen the pupil so the retina can be examined in full detail. Early-stage ROP produces no visible external signs and cannot be detected without this specialist examination. Serial examinations are required to monitor whether the disease progresses, stabilises, or regresses spontaneously.

Yes, untreated ROP can lead to permanent vision loss or blindness. However, the vast majority of cases are preventable when screening occurs at the right time and treatment is started promptly. Stages 1 and 2 often resolve without intervention; the risk of blindness becomes significant from Stage 4 onwards, particularly Stage 5, where total retinal detachment has occurred. Timely diagnosis remains the single most important protective factor.

ROP screening is available at major government hospitals and specialist eye centres across Malaysia. The Ministry of Health Malaysia’s 2023 Clinical Practice Guidelines define which infants qualify — gestational age below 34 weeks or birth weight below 1,750 grams. At OasisEye Specialists, ROP assessments are conducted by paediatric vitreoretinal specialist, Dr Manoharan Shunmugam at the Bangsar South, Kuala Lumpur clinic. Parents should request a referral from their neonatologist or paediatrician if one has not been arranged.

Without intervention, severe ROP can advance to retinal detachment. A Stage 4 or Stage 5 detachment requires complex vitreoretinal surgery — vitrectomy or scleral buckling — and even with successful surgery, some permanent visual impairment is likely. Stage 5, where the retina is completely detached, carries the most guarded visual prognosis. This is why prompt treatment at Stage 3, before detachment occurs, is critical to protecting a child’s long-term sight.

Many children treated successfully for ROP develop good functional vision and attend mainstream school without difficulty. However, children with significant residual vision impairment may benefit from low-vision support, adapted learning materials, or early childhood intervention programmes. Close collaboration between the ophthalmologist, paediatrician, and school is important to ensure any visual challenges are identified and supported early, particularly before formal literacy and numeracy skills are developed.



Medical Disclaimer

This article is written for general educational purposes and does not constitute medical advice. It is not a substitute for professional consultation with a qualified ophthalmologist. Individual clinical situations vary, and management decisions should always be made in partnership with your treating specialist.

Age-related Macular Degeneration (AMD)

Age-related Macular Degeneration (AMD)

Age-related macular degeneration (AMD) is a chronic and painless disease of the macula that can blur your central vision. The macula is part of the retina, which is the light-sensitive tissue at the back of the eye that mainly controls sharp, detailed vision. AMD is a common condition that causes visual loss in those over 50. As we age, the macula can undergo damage, thinning, or abnormal blood vessel growth, leading to vision loss. 

Types

Retinal photographs comparing dry age related macular degeneration and wet age related macular degeneration with visible changes in the macula.

Source:  NEORETINA Eyecare Institute 

There are 2 types of AMD: dry and wet.

Dry AMD, also known as atrophic AMD, affects the majority of AMD patients. It occurs when the light-sensitive cells in the macula slowly break down, gradually blurring central vision in the affected eye.

A less common kind of AMD that typically results in rapid vision loss is wet AMD, also known as neovascular AMD. It occurs when abnormal blood vessels grow in the back of the eye.

Symptoms

  • Difficulty seeing in dim light
  • Blurriness in their central vision
  • Straight lines begin to appear crooked or wavy
  • Hazy patch (blind spot) close to your centre vision
  • Colors appear less bright/saturated 

Side by side comparison of normal vision and vision affected by age related macular degeneration showing a blurred central area.

Source: National Eye Institute, National Institutes of Health

Straight lines looking wavy are a warning sign for late AMD. If you notice this symptom, see your eye doctor right away.

Risk factors

People aged 55 and older are more likely to have AMD. The risk for AMD is also higher for those who:

  • Have a family history of AMD
  • Caucasian
  • Women
  • Smoking
  • Lack of physical activity
  • High blood pressure (hypertension) and high cholesterol levels

Assessment

  • Dilated eye examination. 
  • Amsler chart. This can detect hazy, wavy or missing lines or areas on the grid.

Amsler Grid Test chart used to detect distorted, blurry or missing areas that may indicate macular degeneration.

Source: Pujashah

  • OCT scan. A non-invasive and painless scan for your retina and macula. 

OCT retinal imaging machine used for detailed scanning of the retina and macula in diagnosing macular degeneration.

Treatment

Since there is currently no cure for early AMD, your eye doctor will most likely use routine eye exams to monitor the condition of your eyes. Healthy eating, frequent exercise, and quitting smoking can all be beneficial as well.

Certain dietary supplements, such as vitamins and minerals, may be able to prevent AMD from progressing to the late stage.

On the other hand, if you have wet AMD, there are other treatments that may be able to stop further vision loss:

  • Anti-VEGF injections are the standard treatment involving injections in your eye used to slow the progression of the disease and reduce the damaging effects of these leaky abnormal blood vessels. 
  • Photodynamic therapy (PDT), a combination of injections and laser treatment. A light-sensitive drug is injected into the bloodstream, where it concentrates in abnormal blood vessels below the macula. The drug is then activated by a laser beam that is directed into the eye, resulting in the formation of blood clots, which seal off the abnormal blood vessels. 
  • Laser photocoagulation is used to delay the progression of vision loss by sealing off abnormal blood vessels with a laser. 

Currently, there is no effective treatment to reverse vision loss in late-stage dry AMD, but support is available to help you live with AMD, including help with low vision and lifestyle changes in order to help you have the best quality of life. 

The retinal specialists available in OasisEye Specialists include:

What is Macular Hole?

Example of Macular Hole

A macular hole is a small break or tear in the macula, the part of the retina responsible for sharp, central vision. 

 

How a Macular Hole Forms

The most common cause of macular holes is the aging process. As we age, the vitreous, a gel-like substance in the eye, shrinks and separates. If some of the vitreous remains attached to the retina as it shrinks, it can pull a section of the retina away with it, leading to a hole. This process is known as vitreomacular traction. 

  Cross-section diagram of the eye showing the vitreous body pulling on the retina and a comparison between a normal macula and a macular hole.

Source: Retina Center Chicago

Other causes or risk factors of macular holes include:

 

Symptoms

  • Distorted central vision
  • Blurred central vision
  • Dark or blind spot at central vision
  • Difficulty with tasks requiring fine detail, such as recognizing faces or reading small print

Amsler grid examples comparing normal central vision, a missing central area representing a blind spot, and wavy line distortion caused by a macular hole.

Source: Pure Optical

 

Diagnosis

An ophthalmologist can diagnose a macular hole through several tests, including:

  • Dilated eye examination
  • Optical coherence tomography (OCT): This non-invasive imaging test provides detailed cross-sections of the retina and can help identify the size and location of the hole.

Side-by-side images of a normal retina and a retina with a macular hole, including OCT scans showing the structural difference in the macula.

Source: NERA

 

Treatment

In many cases, surgical intervention is required to repair a macular hole, especially if it affects vision significantly. Vitrectomy is the procedure in which the surgeon removes the vitreous gel and replaces it with a gas bubble. The patient is asked to maintain a specific head position for a period to ensure the gas bubble presses against the hole, aiding in its closure.

 

Conclusion

It’s important for anyone experiencing symptoms like blurred vision, distorted vision, or central blind spots to seek an eye examination.

The vitreoretinal surgeons available in OasisEye Specialists include:

Understanding Uveitis

An eye condition called uveitis.

Uveitis is the inflammation of the uvea (the middle layer of the eye), which includes the iris, ciliary body, and choroid. It can cause eye pain, redness, blurred vision, light sensitivity and floaters.

 

Types of Uveitis

Diagram showing anterior, intermediate and posterior uveitis.

Source: Tennessee Retina

 

Causes of Uveitis

Uveitis can have various causes, which are often classified into the following categories:

1. Autoimmune and Inflammatory Conditions

  • Ankylosing spondylitis
  • Rheumatoid arthritis
  • Juvenile idiopathic arthritis 
  • Sarcoidosis

2. Infections

  • Bacterial, viral, fungal, or parasitic

3. Trauma or Injury

  • Direct eye trauma or injury
  • Secondary to eye surgery

4. Other Cause or Risk Factors 

  • Smoking
  • Cancer related
  • Genetics: certain genetic markers, such as HLA-B27 
  • Idiopathic (no identifiable cause)

 

Symptoms of Uveitis

Symptoms can develop suddenly or gradually and may affect one or both eyes. Common symptoms include:

  • Eye pain (aching or throbbing)
  • Redness in the eye
  • Blurred vision
  • Sensitivity to light (photophobia)
  • Floaters (small dark spots in vision)

 

Diagnosis of Uveitis

  • Dilated Eye Examination: The doctor will use eye drops to widen your pupils, allowing them to check the structures in the back of your eye.
  • Optical Coherence Tomography (OCT): This imaging technique provides detailed cross-sectional images of the retina, showing the separation of retina layers.

                 OCT scan showing cross-section of the retina.Topcon Triton OCT imaging device for eye scans.

  • Widefield Retinal Imaging: It provides a field of view of 200 degrees or 82% of the retina in a single capture. Besides, using special dyes injected into the bloodstream, Fundus Fluorescein Angiography (FFA) or Indocyanine Green Angiography (ICGA) can help to detect blood vessel leakage or fall out which might be caused by uveitis.
    Optos widefield retinal imaging system.
  • Laboratory Investigations: Several examinations including blood test, ESR, PCR test or Chest X-ray could rule out the underlying autoimmune or infectious causes.

    Widefield colour and fluorescein angiography images of the retina.

 

Treatments of Uveitis

The treatment of uveitis depends on the underlying cause, severity, and part of the eye affected (anterior, intermediate, posterior, or panuveitis). Here’s an overview of common treatments:

1. Medications

     A. Corticosteroids (to reduce inflammation)

     B. Immunosuppressive or Biologic Therapy (for non-infectious or autoimmune uveitis)

     C. Antimicrobials (if infection is the cause)

     D. Cycloplegic Drops (to relieve pain and prevent complications)

2. Lifestyle and Supportive Measures

  • Sunglasses – to reduce light sensitivity
  • Regular eye exams – to monitor progress
  • Managing underlying conditions – such as autoimmune diseases

3. Surgery (for severe or refractory cases)

  • Vitrectomy – to remove inflammatory debris or treat complications
  • Cataract or glaucoma surgery – if secondary complications develop

 

Why Early Diagnosis Matters

If left untreated, uveitis can lead to complications such as glaucoma, cataracts, macular edema, or even permanent vision loss. Early diagnosis and prompt treatment are key to preventing serious outcomes.

If you experience persistent eye pain, redness, or changes in your vision, don’t wait—consult an eye care professional.

The medical retina & uveitis specialists available in OasisEye Specialists include Dr Wong Hon Seng who is based in Kuala Lumpur; Dr Rajasudha Sawri Rajan who is based in Kuala Lumpur and Puchong.


FAQs

Yes. If not treated properly, uveitis can lead to complications such as:

  • Glaucoma
  • Cataracts
  • Retinal damage or detachment
  • Vision loss or blindness

Early diagnosis and treatment are critical.

Treatment depends on the cause, severity, and type. Options include:

  • Steroid eye drops (mainstay for anterior uveitis)
  • Oral steroids or injectable steroids
  • Immunosuppressive medications (for autoimmune-related cases)
  • Antibiotics or antivirals (for infectious causes)
  • Mydriatic drops (to dilate the pupil and reduce pain)

Treatment aims to control inflammation, prevent complications, and preserve vision.

Yes, recurrence is common, especially in autoimmune-related uveitis. Long-term monitoring is important even after symptoms resolve.

Most forms of uveitis are not contagious, but if caused by an infection like herpes or tuberculosis, the underlying infection might be.

  • Treat underlying conditions properly (autoimmune diseases, infections)
  • Protect your eyes from injury
  • Attend regular eye exams if you have a history of uveitis or autoimmune disease
  • Follow your doctor’s treatment plan to prevent recurrences

See an eye doctor (ophthalmologist) immediately. Early treatment can prevent serious complications.

Floaters and Flashes: Recognizing Retinal Detachment as an Eye Emergency

Objects blocking vision.

Have you recently noticed sudden flashes of light or floaters in your vision? Are parts of your field of view obscured by a curtain-like shadow? These could be warning signs of retinal detachment, which is a medical emergency condition. 

 

What are the Risk Factors of Retinal Detachment?

  • Eye injury or trauma: For example, a fall, a sports accident, or a car accident.
  • High myopia (short-sightedness): In high myopia individuals, the eyeball is elongated and stretched, resulting in a higher risk of getting a tear or hole in the retina. 
  • Diabetes: High blood sugar levels can damage the blood vessels, causing bleeding or scarring. The scar tissue can pull on the retina and cause detachment.
  • Previous eye surgery: Undergoing certain types of eye surgery may increase the risk of retinal detachment, particularly if the surgery involves the vitreous or the lens of the eye.

 

How is Retinal Detachment Diagnosed?

  • Dilated Eye Examination: The doctor will use special drops to widen your pupils, allowing them to see the retina and other structures in the back of your eye.
  • Optical Coherence Tomography (OCT): This imaging technique provides detailed cross-sectional images of the retina, showing the separation of retina layers.

Optical Coherence Tomography (OCT) machine with scan showing cross-sectional image of detached retina.

  • Widefield Retinal Imaging: It provides a field of view of 200 degrees or 82% of the retina in a single capture. It helps to localise the area and the extent of the detachment.

Optos widefield retinal imaging device with ultra-wide view of retina displaying signs of detachment.

  • Ultrasound: If the retina is not clearly visible (for example, due to bleeding), an ultrasound of the eye may be performed.

Ultrasound imaging device showing retinal detachment on screen.

 

Treatment of Retinal Detachment:

Treating retinal detachment quickly is essential for preserving vision. The treatment approach depends on the type and extent of the detachment.

  1. Laser retinopexy: If a retinal tear or hole is present, a laser can be used to seal it and prevent further detachment.
  2. Cryopexy: It uses extreme cold to seal the tear or hole in the retina.
  3. Pneumatic Retinopexy: A gas bubble is injected into the eye to push the retina back into place, followed by laser or cryotherapy to seal the tear.
  4. Scleral Buckling: A small band (buckle) is placed around the eye to gently push the wall of the eye toward the retina, which helps to reattach it.
  5. Vitrectomy: This involves removing the vitreous gel and replacing it with a gas or silicone oil to help hold the retina in place.

The choice of treatment will depend on the specific circumstances of the detachment, such as its location, size, and the health of the retina.

 

Symptoms of Retinal Detachment: 

  • Floaters (dark spots or lines that seem to move with your vision)
  • Flashes of light
  • A shadow or curtain-like effect in your vision
  • A sudden loss in vision

It’s important to seek medical attention immediately if one experiences the above symptoms.

The vitreoretinal surgeons available in OasisEye Specialists include Dr Kenneth Fong, Dr Manoharan, Dr Wilson Wong who is based in Kuala Lumpur; Dr Ling Kiet Phang who is based in Johor Bahru; Dr Teh Wee Min who is based in Seremban and also Dato Dr Haslina who is based in Penang.


FAQs

Retinal detachment is a serious eye condition where the retina (the light-sensitive layer at the back of your eye) pulls away from its normal position. This can cause permanent vision loss if not treated promptly.

No, retinal detachment is usually painless, which is why recognizing the visual symptoms early is critical.

Not all cases are preventable, but regular eye check-ups help detect early signs like retinal tears. High-risk patients should have routine retinal screenings.

Retinal detachment is an ocular emergency. Delaying treatment can lead to permanent vision loss. If you experience symptoms, seek immediate care.

A retinal tear is a small rip or hole in the retina, which may lead to fluid passing underneath and causing a retinal detachment. Tears are usually treated early with laser or freezing therapy to prevent detachment.

Understanding Central Serous Chorioretinopathy: An In-Depth Look

Central Serous Chorioretinopathy (CSCR) is a retinal disorder where fluid accumulates beneath the macula, the area of the retina which is responsible for central vision. The fluid leakage comes from a layer of tissue under the retina, called the choroid. It often results in blurry or distorted central vision.

OCT scan showing macular edema and retinal thickening.

The exact cause of CSCR is not fully understood, but it is believed to involve a combination of factors, including disturbances in the choroidal circulation, stress, and steroid use. The condition is typically unilateral (affecting one eye), although it can sometimes affect both eyes.

Who is at Risk for CSCR?

CSCR is most commonly seen in adults between the ages of 30 and 50, with men being more frequently affected than women. Some risk factors include:

Frustrated person showing stress.

  • Stress
  • Steroid Use. Both systemic and topical steroids are believed to alter the permeability of the blood vessels in the choroid, leading to fluid leakage.
  • Underlying Health Conditions. For example, hypertension.
  • Genetics
  • Age and Gender. CSCR is 6 times more commonly found in men, particularly those in their 30s to 50s.

Symptoms of CSCR

CSCR will affect vision and may make reading, driving, or recognizing faces more challenging. 

  • Blurry vision
  • Visual Distortions. Straight lines may appear curved or wavy.
  • Micropsia. Patients may perceive objects smaller than they actually are.
  • Dark Spots in Vision
  • Color Perception Changes

How Is CSCR Diagnosed?

In OasisEye Specialists, diagnosis of CSCR is aided by the following  procedures:

Optical Coherence Tomography (OCT): This imaging technique provides detailed cross-sectional images of the retina, showing if there is any fluid accumulation. OCT angiography (OCTA) is a high resolution imaging technique that can detect vascular abnormalities, especially in chronic CSCR cases.

Optical Coherence Tomography (OCT) machine for retinal imaging.OCT showing fluid accumulation under the retina.

Fundus Fluorescein Angiography (FFA): A dye is injected into the bloodstream, and a special camera captures images of the retina to detect abnormal blood vessel leakage or areas of fluid buildup.

Optos retinal imaging device for eye diagnosis.Fluorescein angiography showing retinal blood vessels.

Indocyanine Green Angiography (ICGA): Similar to FFA but ICGA focuses on detecting the leakage of fluid in the choroid layer.

Optos retinal imaging device for eye diagnosis.Fluorescein angiography revealing abnormal retinal spots.

Amsler Grid Test: Patients may be asked to look at a grid of straight lines. Distorted lines or blind spots can help diagnose visual disturbances caused by CSCR.

Amsler grid showing distortion, a symptom of macular degeneration.

Treatment Options for CSCR

In most cases, CSCR resolves on its own without the need for medical intervention. However, in persistent or recurrent cases, treatment may be necessary to reduce the risk of long-term vision damage. Some treatment options include:

  • Steroid Avoidance.
  • Laser Photocoagulation: This technique could seal the leakage point and hastens the resolution of subretinal fluid.
  • Photodynamic Therapy (PDT): This treatment uses a light-sensitive drug called verteporfin and laser to activate the drug. It can close abnormal blood vessels in the choroid and reduce fluid buildup.
  • Anti-VEGF Therapy: In some cases, drugs that block vascular endothelial growth factor (VEGF), which promotes abnormal blood vessel growth, may be used to reduce fluid accumulation.

Prevention of CSCR

While there is no guaranteed way to prevent CSCR, there are several strategies to reduce risk:

  • Stress Management. Regular exercises are recommended.
  • Avoid Steroids.
  • Maintain a Healthy Lifestyle. For example, a balanced diet. 

Conclusion

Central Serous Chorioretinopathy is a serious yet often manageable eye condition that can impact vision. If you experience symptoms like blurred or distorted vision, it’s important to seek the advice of an eye care professional to ensure early intervention and preserve your vision. 

The Retina Specialists in OasisEye Specialists include Dr Kenneth Fong Choong Sian, Dr Manoharan Shunmugam, Dr Wilson Wong Jun Jie, Dr Wong Hon Seng and Dr Rajasudha Sawri Rajan from Kuala Lumpur; Dr Teh Wee Min from Seremban; Dr Ling Kiet Phang from Johor Bahru; and Dato’ Dr Haslina Binti Mohd Ali (D.S.D.K) from Penang.


FAQs

Most cases are temporary and resolve on their own within 1 to 3 months. However, chronic or recurrent CSCR can lead to permanent vision damage if not managed properly.

Yes. Recurrence is common, especially in those with ongoing stress, steroid use, or poor sleep hygiene. Repeated episodes may lead to vision loss.

Total blindness is very rare, but central vision loss or permanent visual distortion can occur in chronic or untreated cases.

No. CSCR is typically painless. Any pain should be investigated further, as it may indicate a different condition.

CSCR is not directly inherited, but genetic susceptibility may play a role. Having a first-degree relative with CSCR may slightly increase your risk.

What is Ocular Toxoplasmosis?

Toxoplasmosis is an infection caused by the intracellular protozoan parasite known as Toxoplasma gondii. The infection may be acquired or congenital, whereby the foetus acquires it from the mother while in the womb. Most people contract toxoplasmosis by eating raw or undercooked meat, vegetables or dairy products, or by coming into contact with infected cats, contaminated sandboxes or cat litter boxes – all of which have been contaminated with cat faeces. Although most parasite-infected individuals do not exhibit any symptoms, some people may experience flu-like symptoms, such as fever, headache and muscle aches. Infants and those with low immune systems are most frequently affected by serious forms of this disease. Toxoplasmosis during pregnancy may cause miscarriage and birth abnormalities.

Ocular toxoplasmosis occurs in the eye, where Toxoplasma gondii can cause inflammation. Frequently, ocular toxoplasmosis results from the reactivation of a congenital infection, although an acquired infection is now regarded to be more common. The parasite typically affects the retina causing eye pain, blurred vision and possibly permanent damage, including blindness.

Fundus images showing ocular toxoplasmosis with retinal lesions marked by arrows, indicating areas of inflammation and damage in the eye.

What investigations are involved?

Ocular toxoplasmosis can look like other eye diseases that cause inflammation in the back of the eye (posterior and panuveitis). The doctor will look at the signs and symptoms to aid in coming to a diagnosis, which is based on clinical findings. If it’s not clear, a test called polymerase chain reaction (PCR) can be done to look for Toxoplasma DNA in the fluid from the eye and blood samples.
There are two imaging tests called Fundus Fluorescein Angiography (FFA) and Indocyanine Green (ICG) that can be used to visualize blood flow in the eye, which may help diagnose ocular toxoplasmosis.

Management / Treatment

Ocular toxoplasmosis requires personalized care, meaning that treatment will vary from person to person depending on factors such as the location of the infection, the severity of inflammation, and the patient’s immune system. If someone with a healthy immune system gets an ocular infection, it will usually go away on its own. However, if the infection affects certain parts of the eye, treatment may be necessary. People with weakened immune systems, such as those who have had transplants or are HIV-positive, may need long-term treatment.
There are several drugs available to treat ocular toxoplasmosis, and doctors often use a combination of drugs to help patients recover quickly and with minimal damage to their eyes. These drugs can kill the parasite responsible for the infection, but they work best when used together. In some cases, surgery may be necessary to treat complications like retinal detachment, cataract and choroidal or epiretinal neovascular membranes involving the macula.

It’s common for ocular toxoplasmosis to recur after someone has had it before, but relapses are not contagious and do not pose a risk to others. Relapses don’t pose a threat to an unborn child if the mother is pregnant.

Prevention

Preventive measures include thoroughly cleaning and washing fruits and vegetables, cooking meat adequately to destroy any harbored cysts, and avoiding contact with cat litter pans during pregnancy. Women of childbearing age must take adequate contraceptive measures for six months following primary toxoplasmosis infection.

In conclusion, preventing toxoplasmosis is crucial for maintaining healthy eyes, and early diagnosis and treatment are essential for managing the infection. At OasisEye Specialists, we offer comprehensive eye care services, including diagnosis and treatment of uveitis caused by toxoplasmosis. Our experienced team of eye specialists is committed to providing personalized care and innovative treatments to preserve and improve our patients’ vision. Contact us today to schedule a consultation and take the first step towards optimal eye health.

Retinitis Pigmentosa

What is Retinitis Pigmentosa?

Retinitis Pigmentosa, also known as RP, is a group of eye conditions that affect the retina. This condition can alter how the retina responds to light, making it difficult for people with RP to see. Essentially, RP causes changes to the cells in the retina known as photoreceptors, which do not work properly and can result in poor vision. The photoreceptors contain rods and cones cells, with rods being affected first in RP patients, causing difficulty seeing at night and loss of peripheral vision. RP causes a slow loss of vision over time, although most people do not become completely blind. RP usually affects both eyes and is often inherited from parents to their children.

Symptoms of Retinitis Pigmentosa

One of the most common early symptoms that retinitis pigmentosa patients experience is night blindness, which makes it difficult to adjust to darkness and see in low light conditions. Patients may also bump into objects in the dark. As the disease progresses, patients gradually lose their peripheral visual field, making it difficult to see objects to the side. In some cases, patients may also experience a loss of central vision in the later stages of the disease.

What Causes Retinitis Pigmentosa?

In most cases, Retinitis Pigmentosa is caused by changes in genes that control the cells in the retina. RP is usually due to genetic inheritance and can be linked to many genes in our body. It can also occur as part of other genetic conditions, such as Usher syndrome, which causes both vision and hearing loss and can lead to difficulty in balancing.

How is Retinitis Pigmentosa diagnosed?

A comprehensive dilated eye examination is required in order to diagnose Retinitis Pigmentosa. Clinical tests such as visual field, electroretinography (ERG), optical coherence tomography (OCT), and fundus autofluorescence imaging (AF) can also be used to diagnose RP. Genetic testing can also be done to determine the type of RP a patient has, which is important for family planning since RP is a genetic inherited disorder.

Treatment for Retinitis Pigmentosa

Unfortunately, there is currently no cure for Retinitis Pigmentosa. However, low vision aids and visual rehabilitation programs provided by low vision specialists can help RP patients improve their quality of life. Researchers are studying gene and cell therapies as potential future treatments for RP.

If you are experiencing any symptoms related to Retinitis Pigmentosa, we recommend visiting a retinal specialist at our centre. A retinal specialist will be able to determine if any diagnostic tests are necessary to rule out any abnormalities. Additionally, we offer genetic testing at our centre.

Epiretinal Membrane (ERM)

An epiretinal membrane (ERM) is an eye condition where a thin, clear, and transparent fibrous cellular material forms on the surface of the retina. It commonly occurs, affecting the posterior pole of the retina over the macula. The cause is unknown, but some research shows that it can be secondary to trauma, post-intraocular surgery, chronic ocular diseases, and so on.

Optical coherence tomography (OCT) scan of the retina showing epiretinal membrane (ERM) with red arrows indicating areas of retinal surface distortion and thickening.

Symptoms:

ERMs usually cause several symptoms, such as:

  • Difficulty in seeing fine details and recognizing faces when the central part of the retina is affected.
  • Blurred and distorted vision.
  • Straight lines appear wavy.
  • Decreased vision and loss of central vision.
  • Double vision.

Causes:

Epiretinal membranes can occur as a result of the normal aging process in the eyes. It is common in individuals over the age of 50. One reason for this condition is when the vitreous gel peels away from the retina. ERM may also form following retinal or eye surgery.

Diagnosis:

An epiretinal membrane can be diagnosed during a routine eye examination for mild cases where the symptoms do not affect the individual. During the eye examination, an optometrist or ophthalmologist may use a non-invasive imaging technique called Optical Coherence Tomography (OCT) to visualize the layers of the retina. In some patients, an ophthalmologist will request additional tests such as fluorescein angiography to determine any other underlying problem that might have caused this problem. The test involves the use of dye to light up areas in the retina.

Treatment:

In many cases, mild membranes are monitored over time for progression. Aside from surgery, there are no other available treatments for epiretinal membranes. Eyeglasses, contact lenses, and prescription eye drops are generally not effective treatments for epiretinal membrane.

Surgery is considered and recommended when the patient’s vision or perception of visual distortion starts to affect their quality of life, for example, when the vision is worse than 20/40 (6/12). To preserve the anatomic integrity of the retina, the membrane removal must be executed with extreme caution. The surgeon will make a tiny cut around 4 mm and remove the fluid from inside the eye. Then, the surgeon will hold and gently peel the epiretinal membrane from the retina and replace the fluid in the eye. The surgery is performed under local anesthesia.

Therefore, it is advisable to visit an ophthalmologist for a detailed eye examination if you experience any unusual symptoms related to an epiretinal membrane (ERM). Moreover, an eye screening can also be beneficial for the early detection of any retinal disorder.